As part of our "big picture" posts, today we cover the relationship between HRV and training load. In particular, this post is inspired by a recent study by Flatt and Howells, that you can find on Andrew's blog here. This is an important topic as it can be a little confusing to the ones approaching HRV measurements and trying to better understand physiological stress in response to training and lifestyle. The view that training should cause a dip in HRV is somewhat simplistic in my opinion. HRV is a measure of physiological stress, and periods of higher cumulative stress are typically highlighted by a reduction in HRV, but acute (day to day) changes are hardly simply linked to TSS or training intensity, let alone the fact that in a situation of positive adaptation to training, your HRV should increase (or at least be stable) when consistently increasing training load (more on this later). So what is the relation between HRV and training load? Do we expect HRV to decrease after hard efforts? How does this relationship change when we look at elite athletes, with respect to the rest of us? Study findingsThe authors started with the following hypothesis, as Andrew wrote in his blog: "Based on previous studies, we’d expect the weekly LnRMSSD mean (LnRMSSDm) to decrease and the coefficient of variation (LnRMSSDcv) to increase relative to baseline. We’ve observed this in collegiate soccer players and sprint-swimmers." - however, this is not what they found. In untrained and recreational athletes, day to day changes in HRV reflect training load quite well, on average, as we have described and published in the past. However, as we become more fit and more used to training stressors, our ability to cope with them changes, and our physiological responses change accordingly. This means we expect less acute drops. The extreme case here is elite athletes, which are used to high levels of volume and intensity, most likely already for years, and therefore physiological responses at the acute level are often less interesting, which is why baseline changes with respect to normal values should be analyzed instead, so that periods of higher stress or good adaptation can be captured, and changes can be made when necessary. This is also what happened in the study mentioned above, as the players showed no change in HRV throughout two weeks of intensified training load. Even the CV, a measure of how well we are adapting to training, with lower values typically meaning better adaptation (HRV is not jumping around as much on a day to day basis), increased during the first few days, but then went back down while load kept increasing. These are changes normally associated with reduced load, so what happened here? As Andrew states "The discrepancy here appears to be related to how players are tolerating and adapting to the training load. We often assume that increased loads will result in fatigue accumulation and temporary negative responses. However, these elite players demonstrated no reductions in subjective indicators of recovery status during the weeks of increased load. Additionally, there was no significant decrement in running performance (maximum aerobic speed) mid-way through the intensified microcycles. Thus, the preservation of autonomic activity (no change in LnRMSSDm) and less fluctuations (reduced LnRMSSDcv) seem to reflect a postive coping response to the training. In fact, individuals who demonstrated the lowest LnRMSSDcv during week 1 of increased load showed the most favorable changes in running performance (r = -0.74)." CV and detected trend in HRV4Training Pro. How are you coping with your training load?What we can derive from this study, similarly also to what was shown in the recent study by Javaloyes and co-authors (covered here), is that a reduced CV is often associated with coping well with training, and the relation we expect to see is not between training load and HRV, but between HRV and adaptation, which is why these metrics are so important.
After all if all HRV was telling us was the intensity of our workout, there would be no need to measure it, but what we are measuring is how we are responding to that workout or block of workouts, with certain trends (stable or upwards HRV, reduced CV) being indicative of good adaptation, even in periods of very high load. This means also that the opposite trends, reductions in HRV or larger fluctuations in CV are signs of poor adaptation and should trigger changes in training. To learn more about these aspects, check out two of our recent blog posts, one on the big picture, and one on how to use HRV to guide training. This is in line also with what Dan Plews argues in his analysis of HRV data pre-ironman, that you can find here. If you are into podcasts, we discuss some of these aspects at the Scientific Triathlon podcast. While acute changes are informative and can help us make small adjustments following a bad night of sleep, a particularly hard workout, or early sign of sickness, what you should be focusing on is certainly the big picture, aiming for your HRV to be within normal range, and making adjustment aiming at prioritizing recovery should your HRV baseline go below normal values, so that performance can be optimized in the long term, as shown in multiple studies. Comments are closed.
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Marco Altini, founder of HRV4Training Blog Index The Ultimate Guide to HRV 1: Measurement setup 2: Interpreting your data 3: Case studies and practical examples How To 1. Intro to HRV 2. How to use HRV, the basics 3. HRV guided training 4. HRV and training load 5. HRV, strength & power 6. Overview in HRV4Training Pro 7. HRV in team sports HRV Measurements Best Practices 1. Context & Time of the Day 2. Duration 3. Paced breathing 4. Orthostatic Test 5. Slides HRV overview 6. Normal values and historical data 7. HRV features Data Analysis 1a. Acute Changes in HRV (individual level) 1b. Acute Changes in HRV (population level) 1c. Acute Changes in HRV & measurement consistency 1d. Acute Changes in HRV in endurance and power sports 2a. Interpreting HRV Trends 2b. HRV Baseline Trends & CV 3. Tags & Correlations 4. Ectopic beats & motion artifacts 5. HRV4Training Insights 6. HRV4Training & Sports Science 7. HRV & fitness / training load 8. HRV & performance 9. VO2max models 10. Repeated HRV measurements 11. VO2max and performance 12. HR, HRV and performance 13. Training intensity & performance 14. Publication: VO2max & running performance 15. Estimating running performance 16. Coefficient of Variation 17. More on CV and the big picture 18. Case study marathon training 19. Case study injury and lifestyle stress 20. HRV and menstrual cycle 21. Cardiac decoupling 22. FTP, lactate threshold, half and full marathon time estimates 23. Training Monotony Camera & Sensors 1. ECG vs Polar & Mio Alpha 2a. Camera vs Polar 2b. Camera vs Polar iOS10 2c. iPhone 7+ vs Polar 2d. Comparison of PPG sensors 3. Camera measurement guidelines 4. Validation paper 5. Android camera vs Chest strap 6. Scosche Rhythm24 7. Apple Watch 8. CorSense 9. Samsung Galaxy App Features 1. Features and Recovery Points 2. Daily advice 3. HRV4Training insights 4. Sleep tracking 5. Training load analysis 6a. Integration with Strava 6b. Integration with TrainingPeaks 6c. Integration with SportTracks 6d. Integration with Genetrainer 6e. Integration with Apple Health 6f. Integration with Todays Plan 7. Acute HRV changes by sport 8. Remote tags in HRV4T Coach 9. VO2max Estimation 10. Acute stressors analysis 11. Training Polarization 12. Lactate Threshold Estimation 13. Functional Threshold Power(FTP) Estimation for cyclists 14. Aerobic Endurance analysis 15. Intervals Analysis 16. Training Planning 17. Integration with Oura 18. Aerobic efficiency and cardiac decoupling Other 1. HRV normal values 2. HRV normalization by HR 3. HRV 101 |